(1) Ramos-Mondragon R, Edokobi N, Hodges S L, et al. Neonatal Scn1b-null mice have sinoatrial node dysfunction, altered atrial structure, and atrial fibrillation[J]. JCI insight, 2022, 7(10).

(2) Liu Y, Li J, Xu N, et al. Transcription factor Meis1 act as a new regulator of ischemic arrhythmias in mice[J]. Journal of Advanced Research, 2022, 39: 275-289.

(3) Liu Y, Lv H, Tan R, et al. Platelets promote Ang II (Angiotensin II)-induced atrial fibrillation by releasing TGF-β1 (transforming growth factor-β1) and interacting with fibroblasts[J]. Hypertension, 2020, 76(6): 1856-1867.

(4) Zhu C, Rajendran P S, Hanna P, et al. High-resolution structure-function mapping of intact hearts reveals altered sympathetic control of infarct border zones[J]. JCI insight, 2022, 7(3).

(5) Apaijai N, Arinno A, Palee S, et al. High‐saturated fat high‐sugar diet accelerates left‐ventricular dysfunction faster than high‐saturated fat diet alone via increasing oxidative stress and apoptosis in obese‐insulin resistant rats[J]. Molecular nutrition & food research, 2019, 63(2): 1800729.

(6) Demers J, Ton A T, Huynh F, et al. Atrial Electrical Remodeling in Mice With Cardiac‐Specific Overexpression of Angiotensin II Type 1 Receptor[J]. Journal of the American Heart Association, 2022, 11(8): e023974.

(7) Zhang Y, Wang H M, Wang Y Z, et al. Increment of late sodium currents in the left atrial myocytes and its potential contribution to increased susceptibility of atrial fibrillation in castrated male mice[J]. Heart Rhythm, 2017, 14(7): 1073-1080.

(8) Zhang Y, Sun L, Xuan L, et al. Long non-coding RNA CCRR controls cardiac conduction via regulating intercellular coupling[J]. Nature communications, 2018, 9(1): 1-14.